Taoism, The Principle of Wu-Wei Research Paper Example | Topics and Well Written Essays - 2000 words

Taoism, The Principle of Wu-Wei - Research Paper Example This paper will shed more light upon Taoism and the principles of Wu-Wei. Confucianism is a term which is very closely related to Taoism, a lot of can be learned about the Chinese people just by knowing more about Taoism and Confucianism. These are terms which reflect the spirit of the Chinese people and what they are made up of. The ancient Chinese society must be given credit for coining the term Taoism, the ideology and the beliefs of the Han nationality is often portrayed when people talk of Taoism. Ancient people in China used to worship spirits and gods and this was how this term originated. â€Å"Daoism encompasses thought and practice that sometimes are viewed as philosophical, as religious, or as a combination of both.   While modern scholars, especially those in the West, have been preoccupied with classifying Daoist material as either â€Å"philosophical† or religious, historically Daoists themselves have been uninterested in such categories and dichotomies.  Ã ¢â‚¬Å"(IEP.UTM) Taoism has raised a debate for the scholars across Europe; they have been completely discombobulated as to whether Taoism falls under religion or whether it falls under philosophy. Some scholars in Europe say that it should be classified under religion while some others say that it should be classified under philosophy so there is no general consensus when it comes to the classification of Taoism and much debate is going on amongst top scholars in Europe. It is very important to know more about the Han dynasty in order to know more about Taoism, no traces of Taoism were found when the Han dynasty ruled China but the beginning of Taoism can be found in important texts like Daodejing and the Zhuangzi. The Chinese people unanimously believe that a teacher by the name Laozi was mainly responsible for writing Daodejing, he was a very dedicated teacher who also opened a school for the Chinese children and single-handedly wrote Daodejing, it was a great achievement but not too many people know of this incredible achievement. People who followed Taoism were called Taoists and they had some similar beliefs, they understood the connection between nature and their bodies and this again was a great achievement. They also knew that God had not made them immortal and they trotted on the difficult path to become immortal. Taoists also believed in experimenting with their bodies, they found new exercises to keep themselves fit; they were also responsible for discovering interesting and new sexual positions. Chemistry was one subject which fascinated the Taoists and quite a few of them tried new experiments to discover something new which would help them but these experiments did not go down too well and many of them lost their lives trying to do too much. There were emperors who were also fascinated with Chemistry and they met with the same fate when they tried doing the same experiments, this clearly goes to show that there were hungry for success and wanted to find new things which would help their fellow human beings. Self-cultivation was a major objective of Taoism, every Taoist wanted to inculcate this quality in him and pass it on to their family members. â€Å"The teachings that were later called Daoism were first known under the name of Huanglao Dao in the 3rd and 2nd cent. BCE. The thought world transmitted in this stream is what Sima Tan meant by Daojia. The Huanglao School was a center of Taoist practitioners in the state of Qi (modern Shandong). Huangdi was the name for the Yellow

analysis of klinefelters syndrome

analysis of klinefelters syndrome Many chromosomal abnormalities occur early in development and involve the sex chromosomes. Klinefelters Syndrome falls directly into this category. Klinefelters Syndrome is a genetic condition affecting the male population. The following information observes who discovered Klinefelters Syndrome and when it was first diagnosed. The etiology or genetic and environmental factors of the condition are discussed as well as complete description of the disability and the clear diagnostic criteria. The affects of Klinefelters Syndrome are more recognized in adulthood, when it is typically diagnosed. Many Klinefelters patients live out their entire lives without ever knowing they have the condition. Upon conclusion, there should be an adequate amount of information that will give you, the reader, valuable knowledge into the diagnosis and treatment of Klinefelters Syndrome. History Klinefelters Syndrome was first diagnosed in 1942 at the Massachusetts General Hospital in Boston Massachusetts (Schoenstadt, 2006). Dr. Harry Klinefelter was completing his fellowship at the Massachusetts General Hospital when he was assigned to work with Dr. Fuller Albright, also known as the father of endocrinology (Bock, 1993; Klinefelter Syndrome, 2006). Dr. Klinefelter came to examine nine adult men that had a common set of symptoms during the course of his fellowship (Klinefelter Syndrome, 2006). Dr. Klinefelter organized a case study that involved these nine men and their similarities and was encouraged by Dr. Albright to lead the study (Klinefelter Synrome, 2006). The Journal of Clinical Endocrinology was published in November of 1942 with the completed case study of these nine mens similar qualities, which Dr. Klinefelter identified as Klinefelters Syndrome (Klinefelter Syndrome, 2006). The report written by Dr. Klinefelter on these men described them as having testicular dysgenesis, elevated urinary gonadotropins, eunuchoidism, azoospermia, and gynecomastia, all of which have an effect on the underdeveloped size of the testes, the lack of the amount of testosterone produced by the testes, and infertility (Schoenstadt, 2006; Visootsak Graham, 2006). These adult males also exhibit enlarged breast and sparse facial hair (Schoenstadt, 2006). Two groups found out fourteen years after Dr. Klinefelters original description of the syndrome, that the buccal mucosal cells contained an extra chromatin mass or that the cells were chromatin positive (Klinefelter, 1966). Although the patients were described as having a positive female sex chromatin, Dr. Klinefelter states that the patients are phenotypic males and should never be considered otherwise (Klinefelter, 1996). Fourteen years after Dr. Klinefelter first diagnosed Klinefelters Syndrome, another case study was performed to further understand the characteristics that define the condition (Klinefelter Syndrome, 2006). In 1956, Dr. Joe Hin Tjio and Dr. Albert Lavan took the research further to determine the factors that made those men with Klinefelters Syndrome dissimilar from normal adult males through genetic research (Klinefelter Syndrome, 2006; Schoenstadt, 2006). With the advanced experimental methodology available, Dr. Joe Hin Tjio and Dr. Albert Lavan found that humans had 23 pairs of chromosomes, confirming 46 chromosomes, which prior to this time there was thought to be 48 chromosomes (Bock, 1993; Klinefelter Syndrome, 2006). This clarification by Dr. Tjio and Dr. Lavan is the basic foundation for modern cytogenetics, the study of chromosomes and diseases originating from numerical or structural abnormalities in chromosomes (Klinefelter Syndrome, 2006). Dr. Tjio and Dr. Lavan discovered that men with symptoms of Klinefelters Syndrome had an extra sex chromosome that created the chromosomal arrangement of XXY, which is distinct to the normal male chromosomal arrangement of XY (Klinefelter Syndrome, 2006; Schoenstadt, 2006,). Klinefelter Syndrome was still believed to be an endocrine disorder of unknown etiology at this time (Visootsak Graham, 2006). In 1959, just three years after Dr. Tjio and Dr. Lavan made their historical discoveries, an English researcher by the name of Dr. Patricia Jacobs and her associate Dr. J. A. Strong published a study supplementing earlier studies (Klinefelter Syndrome, 2006; Noble, 2003). Dr. Jacobs and Dr. Strong found the link between the endocrinal disease and the extra X sex chromosome (Noble, 2003). Dr. Jacobs linked forty-seven chromosomes in Klinefelters Syndrome males and determined it to be the X chromosome, which she considered an aneuploidy defined as an unusual number of chromosomes and labeled 47, XXY (Bock, 1993; Klinefelter Syndrome, 2006; Schoenstadt, 2006). The 1970s brought forth a larger examination of males born with Klinefelters Syndrome (Bock, 1993). During this time doctors began screening newborn male babies for the extra chromosome (Bock, 1993; Visootsak Graham, 2006). The most significant of the studies done at this time was sponsored by the National Institute of Child Health and Human Development (NICHD) whom examined over forty thousand infants for this extra chromosome (Bock, 1993; Visootsak Graham, 2006). This study was important for the reason that most studies done prior to the 1970s were biased and primarily done on adult males in mental institutions and the penal system (Visootsak Graham, 2006). At this time is when the prevalence of Klinefelters Syndrome was noticed as frequently as one in five hundred to one in one thousand male newborns (Bock, 1993; Visootsak Graham, 2006). Also observed in this study was the reduction in speech and language abilities as well as decreased reading and spelling achievement (Bock, 1993; Visootsak Graham, 2006). Along with these disabilities, Klinefelters patients are characterized by an increased tendency towards fertility, endocrinal, and psychiatric disorders (Noble, 2003). This study demonstrated that most but not all of these males born with the extra chromosome will have these characteristics, and many demonstrate varying degrees of the characteristics (Bock, 1993; Visootsak Graham, 2006). Based on this research it has been found that the extra X chromosome that causes Klinefelters Syndrome is very common, however, the symptoms and characteristics that are most recognizable are quit uncommon (B ock, 1993). Most males are not diagnosed as having Klinefelters Syndrome until they reach adulthood, and many that have the syndrome are never diagnosed as having this chromosomal defect at all (Bock, 1993). One pediatrician at the University of Colorado Medical School in Denver and the director of the National Institute of Child Health and Human Development (NICHD) during the major screening research referred to these newborn males as not having Klinefelters Syndrome because of the possibility that the characteristics may not develop into a syndrome (Bock, 1993; Visootsak Graham, 2006). Etiology and Genetic Factors Every normal human cell has 46 chromosomes that are made up of 23 pairs (Stewart, 2007). Of these 23 pairs, there are 22 that are exactly the same in both males and females called autosomes (U.S. National Library of Medicine, 2010). The 23rd pair of sex chromosomes is what makes males and females different in that the male will have only one X and Y chromosome whereas the female will have two copies of the X chromosome (Stewart, 2007; U.S. National Library of Medicine, 2010). During the formation of the egg and the sperm, or gametes, the chromosomes are halved through a process called meiosis (Stewart, 2007; The Dorsey, 2009). Cells that carry a single chromosome such as the X or Y chromosome are called haploid cells (The Dorsey, 2009). When the egg and sperm join carrying 23 chromosomes each they create the fertile egg, or zygote, which has two haploid sets of chromosomes (The Dorsey, 2009). Therefore, the baby receives two copies of each chromosome, 46 total chromosomes, just like the parents (Stewart, 2007). The extra X in Klinefelter Syndrome is caused from either nondisjunction or anaphase lag. Nondisjunction occurs when the chromosome pairs do not separate as they are intended in the meiosis I or meiosis II stage (Pineyard Zipf, 2003; Stewart, 2007). When this happens there may be a chromosome pair with 24 chromosomes instead of the 23 chromosomes (Stewart, 2007). If this chromosome pair of 24 joins with an egg or sperm with 23 chromosomes then it results in a karotype with 47 chromosomes (Stewart, 2007). In this case there will be three copies of chromosomes rather than the usual two copies of chromosomes (Stewart, 2007). The sperm or egg may donate the extra X chromosome at conception causing a chromosomal abnormality (Mayo Foundation for Medical Education and Research, 2008; Stewart, 2007). This forms the XXY chromosomal formation, which is diagnosed as Klinefelters Syndrome. At least half of 47, XXY conceptions are spontaneously aborted (Pineyard Zipf, 2003). The chromosomal abnormality is random and not known to be caused by any environmental factors (Genetic Science Learning Center, 2010; Mayo Foundation for Medical Education and Research, 2008; National Institute of Health, 2007). This anomaly happens entirely by chance and is unrelated to family history prior to the male childs birth (Mayo Foundation for Medical Education and Research, 2008). This is to say that the male embryos likelihood of being born with Klinefelters Syndrome is not increased or decreased by what the parent does or does not do (Mayo Foundation for Medical Education and Research, 2008). Klinefelters Syndrome is not affected by race (Chen, 2010). This is a completely random occurrence of the sex chromosomes not successfully separating during the formation of the egg or the sperm (Genetic Science Learning Center, 2010). Once this occurs the extra chromosome is then copied into every cell of the embryo (Genetics Science Learning Center, 2008). There are extremely rare cases when there may be three or four extra X chromosomes in all copies of the cells known as 48,XXXY or 49, XXXXY (Stewart, 2007). The 49, XXXXY mosaic is also known as Fraccaros Syndrome and is the most rare form of Klinefelters Syndrome (Duenas et al., 2007). This rare chromosomal abnormality results in more exaggerated features of Klinefelters Syndrome (Stewart, 2007). There are instances where an extra X chromosome is found in only some of the cells (Stewart, 2007). This can be found as two different chromosomal patterns (Stewart, 2007). One pattern occurs when some cells have 46 chromosomes and some have 47 chromosomes (Stewart, 2007). The other pattern is called the mosaic XXY syndrome, or chromosomal mosaicism, and affects approximately six percent of these cases, with the most rare cases being the 48, XXXY or the 49, XXXXY, or other arrangements of X chromosomes (Stewart, 2007). The mosaic XXY syndrome occurs only after conception from a mistake in cell division (Stewart, 2007). Anaphase lag is a result of a gamete lacking a sex chromosome (Klinefelter, 1966). When this chromosome lags it is not incorporated into the new cell during the mitosis stage (Kinefelter, 1996). Anaphase lag is thought to be a reason for the mosaic variations of Klinefelters Syndrome (Klinefelter, 1966). Although the chromosomal abnormality of 49, XXXXY is considered to be a variant form of Klinefelters Syndrome, it appears to have a very independent, distinct phenotype (Duenas et al., 2007). Males that show the 49, XXXXY chromosomal structure have much more severe clinical features than that of a Klinefelters Syndrome male (Duenas et al., 2007). This is the most rare of the Klinefelters Syndrome variants and has been reported in over one hundred cases with the frequency being approximately 1 in 85,000 newborn males (Duenas et al., 2007). There have been reports of an even more extreme variant of Klinefelters Syndrome mosaic in newborn males (Duenas et al., 2007). This variant is a 47, XXY/48, XXXY/49, XXXXY mosaicism and has only been reported in three cases according to a researcher in Mexico (Duenas et al., 2007). This means that the male newborn would have the whole spectrum of XY variations. Another variant that affects only males is the 46, XX chromosomal variation (Bock, 1993). This condition occurs when individuals have two X chromosomes in each cell, but are male in appearance. These individuals have male external geniltalia. These individuals also have small, undescended testes possibly along with an urethra opening on the underside of the penis. A small amount of 46, XX Males have external geniltalia that dont clearly resemble either male or female genitalia. These individuals are typically raised male. Phenotypically, there are three groups of these sex-reversed individuals. The first group includes phenotypically normal XX Males, the second group includes the males with genital ambiguities, and the third group is the true hermaphrodites (Bock, 1993). Description of Characteristics or Traits Klinefelters Syndrome has only one constant physical description and that is the small testicular size (Visootsak Graham, 2003). Boys with Klinefelters Syndrome have variable phenotypic characteristics with no obvious facial dysmorphology (Visootsak Graham, 2003). The presence of gynconemastia, or enlarged breast, and other findings of eunuchoid body habits and sparse body hair vary (Visootsak Graham, 2003). Eunuchoid or eunuchoidism is defined as an abnormal condition in males, characterized by underdeveloped reproductive organs with some female characteristics, such as a higher voice or the lack of facial and body hair that results in the lack of male sex hormones (Eunuchoidism, n.d.). Gonadotropins are produced by glands, such as the pituitary, and can result in sparse body hair when not produced adequately (Gonadotropin, 2010). The medical dictionary states that eunuchoidism is marked by a deficiency of sexual development with the persistence of prepubertal characteristics, and often has the presence of characteristics that are typical of the opposite sex (Eunuchoidism, n.d.). Another likely characteristic is azoospermia (Schoenstadt, 2006; Visootsak Graham, 2006). Azoospermia is defined as having little or no sperm count (Azoospermia, 2010). Testicular dysgenesis, or gonadal dysgenesis, is another characteristic of Klinefelters Syndrome (Schoenstadt, 2006; Visootsak Graham, 2006). Testicular dysgenesis is considered a reproductive system developmental disorder that causes a progressive loss of primordial germ cells, or cells that create gametes, in the developing gonads of an embryo (Gonadal dysgenesis, 2010). This gonadal dysgenesis can lead to the extremely hypoplastic, or underdeveloped, and disfunctioning gonads mainly composed of fibrous tissues (Gonadal dysgenesis, 2010). Most infants and children with the 47, XXXY chromosomal abnormalities go through normal growth stages. It is not until puberty that the Klinefelters Syndrome characteristics or traits become more prevalent and noticeable (Visootsak Graham, 2003). There is a significant increase in height between the ages of five and eight (Visootsak Graham, 2003). Another characteristic of Klinefelters Syndrome is the elongated length of arms and legs (Klinefelter, 1966). There is a decrease in androgen production that causes the secondary sexual characteristics to not fully develop (Visootsak Graham, 2003). An androgen is any substance such as androsterone or testosterone that supports male characteristics (Androgen, n.d.). Typically Klinefelters males are infertile (Visootsak Graham, 2003). However, there have been cases of impregnation without the assistance of medical technology (Visootsak Graham, 2003). Autoimmune diseases such as juvenile arthritis can also be present in Klinefelters adolescents. Whereas boys with Klinefelters Syndrome are generally tall with long limbs and remain thin until puberty, they tend to suffer from obesity latter in life. Neurocognitive effects of Klinefelters Syndrome may be more subtle than that of the physical stigmata. Klinefelters males have been found to have relative deficits on verbal IQ subtests and have verbal IQ scores around 20 points lower than those of unaffected siblings. There are also deficits in articulation, word finding, phonemic processing, verbal memory, language comprehension, oral expression problems, as well as linguistic processing speed. It seems that the speech/language problems and some motor deficits are most common in Klinefelters males that have an extra X chromosome. Ninety-two percent of individuals with Klinefelters Syndrome confirm difficulty learning to read. Seventy percent had reading achievement discrepancies or absolute reading deficits on standardized testing. A group of boys with mental retardation and suspicion of fragile X were subject to a genetic screening and the results showed that eight of these boys had Klinefelters Syndrome. Most of the more extreme verbal, visuospatial, and motor skills, such as found in mental retardation and fragile X syndrome are typically spa red. However, some boys with Klinefelters Syndrome suffer from poor manual dexterity and are commonly found to be clumsy and below average in sports (Wodrich Tarbox, 2008). There are many different factors that may underlie linguistic and reading problems. One possibility is a dysfunction of the left hemisphere that may be related to diminished gray matter or a lack of hemispheric asymmetry, or both. It is also possible that executive and frontal deficits may be a cause (Wodrich Tarbox). There is evidence that language is a fundamental issue for Klinefelters children and this can result in further scholastic issues. This problem seems to manifest as dyslexia as defined by poor reading in the setting of normal intelligence. Klinefelters males have also been observed to have difficulties with arithmetical functions. The deficits in auditory processing and verbal memory are the two key cognitive processes that underlie these difficulties. These deficits are also true for normal chromosomal children with dyslexia. The findings are supportive of the concept that defects in frontal systems seem to be caused by a language-based, left frontal-systems problem (Geschwind Dykens, 2004). Adult Klinefelters males have reported to have difficulties with mental flexibility (Wodrich Dykens, 2004). Even with these studies, it should be noted that not all adults that have Klinefelters Syndrome show these classic patterns of verbal deficits that are observed in children (Geschwind Dykens, 2004). However, these findings are not appropriate for all Klinefelters males, many of which complete high school and move on to post-secondary education successfully (Wodrich Tarbox, 2008). Two characteristics that has been falsely associated with Klinefelters males in the past, is sociopathy and criminal behavior. There is, contrary to this belief, fewer psychiatric problems reported among these individuals. However, there are commonly traits of introversion, unassertiveness, and a paucity or lack of ambition. There are also possible traits of impulsivity and social inappropriateness (Wodrich Tarbox, 2008). A Reiss Profile of Fundamental Goals measurement was used to assess the degrees of which Klinefelters males were motivated in 15 domains (Geschwind Dykens, 2004). The Reiss Profile generates a profile that is based on the motivational sensitivities across the domains of aversive sensations, citizenship, family, curiosity, honor, independence, food, order, physical exercise, rejection, power, sex, social contact, vengeance, and social prestige. The Reiss Profile is a well-established psychometric measure that is being used more and more to assess people with and without mental retardation (Geschwind Dykes, 2004). The results suggested that the Klinefelters male group was not particularly motivated by the need for social prestige, independence, or the desire to seek vengeance. This group was also not motivated to avoid physical pain. The general motivator for all the Klinefelters males in this group was curiosity. There were no age effects to this study (Geschwind Dykes, 2004). Characteristics in Adulthood There is a persistent deficiency of androgen in adulthood that can result in the loss of libido, decreased muscle bulk and tone, decreased bone density, a propensity for thromboembolism (an obstruction in a vein or artery from a blood clot), and an increased risk of mortality from cardiovascular and diabetic complications. A common characteristic for Klinefelters adults is gynecomastia (Wattendorf Muenke, 2005). Gynecomastia involves the risk of developing breast carcinoma. There is 200 times more of a risk for Klinefelters males to develop breast carcinoma than other karyotypically normal individuals. This may be a result of the estradiol (the prominant sex hormone in females) to testosterone ratio being so much higher that karyotypically normal men. Another possibility is that it is caused by the increase of peripheral conversion of testosterone to estradiol (Visootsak Graham, 2006). There are different views as to whether Klinefelters adult males are more aggressive or have a greater chance of psychological issues depending on the resource. One study describes the differences as relative to individual testosterone levels and the age at which they received the diagnosis (Morris, Jackson, Hancock, 2009). Equally, there is an impact from the way the diagnosis is reacted to by the Klinefelters male, the family, and friends or peers. The seven major themes that emerged from this study were the diagnosis, the testosterone treatments, health care problems, appearance, self-identity, relationships, and school and education. Of the Klinefelters adults studied, 60 % reported clinical levels of anxiety and 34% had clinical levels of depression. The results of this study show that a prolonged lack of testosterone can have far reaching negative effects on the Klinefelters adult (Morris, Jackson, Hancock, 2009). The historical studies show a disturbingly increased risk for psychiatric disturbance, criminality, and mental retardation. However, these results are outdated and extremely questionable given the initial examinations were given to institutionalized populations (Chen, 2010). Differential Diagnosis Classic Klinefelters Syndrome, 47, XXY, cases make up approximately 80-90% of all Klinefelters diagnosis. There are approximately 6-10% of these cases that are mosaics, which are the cells with 46, XY/47, XXY; 46, XY/48, XXXY; and 47, XXXY/48, XXXY (Chen, 2010; Visootsak Graham, 2003; Visootsak Graham, 2006). In 5% of the cases there are two X chromosomes without a Y chromosome or 46, XX (Visootsak Graham, 2006). The other cases were karyotypes 48, XXXY, 48, XXYY, 49, XXXXY, and 49, XXXYY (Visootsak Graham, 2003). Approximately 1% of these cases are due to a structurally abnormal X with a normal X and Y chromosome described as kayotypes 47, X,i(Xq)Y and 47, X,del(X)Y (Chen, 2010). Klinefelters Syndrome variants occur much less frequently than the classic 47, XXY chromosomal abnormality (Bock, 1993; Visootsak Graham, 2006). Klinefelter variant 48, XXXY is characterized by being average or tall stature with ocular hyperterlorism, which are widely spaced or deep set eyes; flat nasal bridge; curving of the fifth finger, or clinodactyly. Other characteristics are small penis and testicles with hypergonadotropic hypogonadism, which is the absence or decrease in function of the male testes. Theses individuals intelligence quotients range from 40-60. Variant 48, XXYY is characterized by having a tall stature, an eunuchoid habitus with long legs, sparse body hair, small testicles and penis, hypergonadotropic hypogonadism and gynecomastia. These individuals intelligence quotients range from 60-80. Males with variant 49, XXXXY are severely affected. They have smaller than average head circumference also known as microcephaly, short stature with ocular hypertelorism, flat nasal bridge, and upslanting palpebral fissures. Cleft palates are present along with small geniltalia and a heart defect known as patent ductus arteriosus. These individuals intelligence quotients range from 20-60. (Visootsak and Graham, 2003). Klinefelters Syndrome 47, XXY, has no major physical signs, which explains why it may go undiagnosed or misdiagnosed throughout an individuals life. Also with no physical signs, it is truly only diagnosed when genetic testing occurs for a variety of unrelated reasons. Klinefelters Syndrome may be diagnosed prenatally or during early childhood, as an adolescent during puberty, or as an adult when there are recognized fertility problems (Bock, 1993). Klinefelters Syndrome can be diagnosed prenatally through amniocentesis or chorionic villus sampling (Bock, 1993). These tests are normally done if the pregnant woman is older than 35, if there is a family history of genetic defects, or when other medical indications exist (Bock, 1993). A pediatrician may suspect a male child as having Klinefelters Syndrome if there are delays in learning to talk or difficulties in reading and writing as well as physical abnormalities during adolescence (Bock, 1993). Treatments and Interventions All hope is not lost when it comes to the treatment and interventions of the undesirable traits and characteristics that males diagnosed with Klinefelters Syndrome may display or develop. It is recommended that Klinefelters males have a comprehensive neurodevelopmental evaluation as soon as they have been diagnosed. A multidisciplinary developmental evaluation can determine the appropriate treatments during infancy and early childhood. These treatments may include physical therapy, infant simulation programs, and speech therapy (Wattendorf, 2005). If the language difficulties are detected in childhood, then there is more of a possibility for intervention. The language barriers that Klinefelters males may have to cope with can not only affect their academics, it can obstruct their building of social relationships and learning social skills necessary for these relationships. Here is where the Klinefelters child could benefit from a social skills training program. In a social skills training program, the Klinefelters child will be able to practice talking and listening, observing childrens making friends processes, sharing of information, attitudes, and beliefs. This will also assist them in proper classroom behavior and playground behavior. Language disabilities and barriers can prevent Klinefelters males from fitting in socially, so this kind of intervention and assistance can benefit the child greatly. Hearing can be an issue if frequent ear infections occur. Hearing test and screens should be done to ensure that a hearing impairment is not a part of the language difficulties. If the Klinefelters child is not communicating effectively with single words by the ages of 18 to 24 months, then consultation with a speech and language pathologist will be very beneficial (Klinefelter Syndrome Information, 2002). Teachers should be informed of the difficulties that a Klinefelters child may be dealing with in the classroom. A teacher may consider the Klinefelters child to be lazy and daydreaming and a teacher may even forget the child is even in the room. This can result in the Klinefelters child falling behind and eventually being held back a grade. Under the Public Law 94-142, the Individuals with Disabilities Education Act, adopted by Congress in 1975, all children with disabilities have a right to a free, and appropriate public education (Klinefelter Syndrome Information, 2002). Once the Klinefelters male reaches puberty there is usually an inability to produce a normal amount of testosterone. This along with hypogonadism can result in impaired bone mineral density and skeletal muscle development. Also associated with testosterone deficiency is a decrease in libido and energy (Wattendorf Muenke, 2005). Androgen therapy or Testosterone Treatment should begin by time the Klinefelters male reaches middle school, approximately 12 to 14 years of age, based on the level of pubertal development (Klinefelter Syndrome Information, 2002; Wattendorf Muenke, 2005). Testosterone Treatment will ultimately increase the muscle size and strength, as well as, promoting the growth of body and facial hair. It must be noted that Testosterone Treatments can also bring on psychological changes. It is important to adequately inform the parent(s) and the child of these changes so that they can make the most informed decision (Klinefelter Syndrome Information, 2002). There are diff erent ways to receive Testosterone Treatment and that is through injections, transdermal (patches, gels, or creams), orally, or implantation. The kind of testosterone injection will depend mainly on the dosage used and the country in which you receive the injections. Some injectable testosterone esters are Testosterone enanthate, Testosterone cypionate, Sustanon, Testosterone propionate, Testosterone phenylpropionate, Omnadren, and Aqueous testosterone suspension. Types of transdermal patches are Androderm and Testosterone TTS. Two different kinds of testosterone gels and creams are Androgel, and Testim. A few oral supplements include Methyltestosterone and Testosterone undecanoate. The last form of Testosterone Treatment is the Subcutaneous testosterone pellet, which is delivered by implanting a pellet of pure, crystalline testosterone under the skin of the buttocks or abdomen (Testosterone Types and Delivery, n.d.). Adult males with Klinefelters Syndrome usually develop gynecomastia which predisposes men to breast cancer. Therefore, it is important that Klinefelters males do monthly breast examinations. If gynecomastia causes psychological or physical problems, then possible treatment would be cosmetic surgery to remove the breast tissue (Wattendorf Muenke, 2005). Swerdlow et. al (2005) stated that men with Klinefelter Syndrome have elevated risks of several cancers. Prostate cancer, along with breast cancer was more prevalent. Men with Klinefelter Syndrome are also at a substantially higher risk for non-Hodgkin lymphoma, and possibly lung cancer. Breast cancer risk is higher in 47, XXY mosaics. Adult males may face possible infertility issues due to the lack of testosterone production, but if diagnosed early on, this can be minimized and they will be able to reproduce without outside assistance. Summary Klinefelter Syndrome is one of the more recently discovered medical syndromes. Klinefelter Syndrome is not one that causes major dysfunctions and is usually only discovered during genetic testing for infertility or during prenatal testing due to maternal age or prior genetic issues within the family. Because Klinefelter Syndrome has not had a lot of research until the last few years, there is no federal funding set aside for this syndrome. Families with sons that are found to be affected by it have no real support system that is knowledgeable of this syndrome and have to research on their own and create resources to fit their situation as none are available in most areas.

Placental Ruminants and Herbivorous Marsupials of Australia Essay

Placental Ruminants and Herbivorous Marsupials of Australia The marsupial animal species that have evolved on the isolated continent of Australia are unique compared to the rest of the animal kingdom in many ways due to the harsh and distinctive environment found on the continent. The major area of marsupial biology that distinguishes them from all other eutherian mammals is their mode of reproduction. However, it can be said that there are many other areas in which unique differences can be seen between marsupials and eutherian mammals; one such area of adaptation is in the anatomy and physiology of digestion, which distinguishes them from many other similar animals worldwide. The foregut fermenters, such as kangaroos and wallabies, evolved in regions of poor forage quality to be able to extract the most nutrients out of the poorest feeds. Fermentation in the foregut has many advantages over hindgut fermentation, which is seen in marsupials such as the koalas and wombats. The anatomy and physiology of the digestive tracts of both types of fermenters, however, are uniquely suited to their individual modes of nutrition. It can be suggested that the foregut fermenters of the marsupials are very similar to the eutherian ruminants, as both types of adaptations are designed to increase fiber digestibility and increase nutrient absorption. However, there are many major differences between the two groups. Ruminants have developed a four-chambered stomach system, which is not seen in the marsupials, as well as a rumination cycle which allows for the rechewing of previously ingested meals. While regurgitation may be evident in marsupials, it is, however, not analogous to rumination, and is instead called â€Å"merycism.†... .... 23 March 2004. Milewski, A.V. and R.E. Diamond. 2000. Why are very large herbivores absent from Australia? A new theory of micronutrients. Journal of Biogeography. 27(4): 957-978. Russell, J.B. 1988. â€Å"Ecology of Rumen Microorganisms: Energy Use.† In Dobson, Alan, and Marjorie J. Dobson, eds. Aspects of Digestive Physiology in Ruminants: Proceedings of a Satellite Symposium of the 30th International Congress of the International Union of Physiological Sciences. Cornell University Press: Ithaca, NY, pg 74-98. Stevens, C.E. and I.D. Hume. 1995. Comparative Physiology of the Vertebrate Digestive System. Cambridge: Cambridge University Press. Strahan, Ronald, ed. 1995. The mammals of Australia. Smithsonian Institution Press: Washington, DC. Van Soest, Peter J. 1994. Nutritional ecology of the ruminant. Ithaca: Comstock Publishing. Placental Ruminants and Herbivorous Marsupials of Australia Essay Placental Ruminants and Herbivorous Marsupials of Australia The marsupial animal species that have evolved on the isolated continent of Australia are unique compared to the rest of the animal kingdom in many ways due to the harsh and distinctive environment found on the continent. The major area of marsupial biology that distinguishes them from all other eutherian mammals is their mode of reproduction. However, it can be said that there are many other areas in which unique differences can be seen between marsupials and eutherian mammals; one such area of adaptation is in the anatomy and physiology of digestion, which distinguishes them from many other similar animals worldwide. The foregut fermenters, such as kangaroos and wallabies, evolved in regions of poor forage quality to be able to extract the most nutrients out of the poorest feeds. Fermentation in the foregut has many advantages over hindgut fermentation, which is seen in marsupials such as the koalas and wombats. The anatomy and physiology of the digestive tracts of both types of fermenters, however, are uniquely suited to their individual modes of nutrition. It can be suggested that the foregut fermenters of the marsupials are very similar to the eutherian ruminants, as both types of adaptations are designed to increase fiber digestibility and increase nutrient absorption. However, there are many major differences between the two groups. Ruminants have developed a four-chambered stomach system, which is not seen in the marsupials, as well as a rumination cycle which allows for the rechewing of previously ingested meals. While regurgitation may be evident in marsupials, it is, however, not analogous to rumination, and is instead called â€Å"merycism.†... .... 23 March 2004. Milewski, A.V. and R.E. Diamond. 2000. Why are very large herbivores absent from Australia? A new theory of micronutrients. Journal of Biogeography. 27(4): 957-978. Russell, J.B. 1988. â€Å"Ecology of Rumen Microorganisms: Energy Use.† In Dobson, Alan, and Marjorie J. Dobson, eds. Aspects of Digestive Physiology in Ruminants: Proceedings of a Satellite Symposium of the 30th International Congress of the International Union of Physiological Sciences. Cornell University Press: Ithaca, NY, pg 74-98. Stevens, C.E. and I.D. Hume. 1995. Comparative Physiology of the Vertebrate Digestive System. Cambridge: Cambridge University Press. Strahan, Ronald, ed. 1995. The mammals of Australia. Smithsonian Institution Press: Washington, DC. Van Soest, Peter J. 1994. Nutritional ecology of the ruminant. Ithaca: Comstock Publishing.

Citizens Have to a Guaranteed Minimum Income in a Democratic Society Essay

â€Å"Although abuse of the system are inevitable, social welfare payments are essential to protect the rights citizens have to a guaranteed minimum income in a democratic society† Discuss. Social welfare is an essential element of an advanced society. Good systems are always abused, but that does not mean they are faulty. In my opinion, the two main reasons why welfare payments are necessary are as follows: First of all, critics forget that there are many forms of welfare besides payments to the unemployed. Their negative opinions harm those who are not capable of earning a wage, such as single-parent mothers, the disabled, and the sick. Moreover, the unemployed have the right to an income, too. They are not always at fault for not having a job, and in most cases the tax they have paid in the past entitles them to assistance. The second reason is that crime increases when people have no means of support. The desperately poor inevitably turn to crime, which is not only dangerous but costly. Policing the streets is more expensive than providing welfare. A policeman’s wage is four or five times higher than a â€Å"dole† payment. Certain members of society believe that people should look after themselves. They point out that welfare increases dependency on others and destroys dignity. This may be true, but in the case of the unemployed, the relief payments are usually temporary. It is surely the fault of the government if there are long-term unemployed. Welfare critics also believe that it is the responsibility of a victim’s family to provide financial assistance. However, it is too expensive to provide complete help for a severely disabled person. To conclude, it is vital to understand the need for welfare in a modern democratic society. Without welfare payments the poor are destined to become poorer. The first duty of a government is to provide a financial safety net for all disadvantaged persons, and that includes those without work.

Choices based on appearances of their options Essay

Often times people make choices based on appearances of their options- the result of the choice tends to be strongly negative and it is hard to admit a mistake has been done. Often times things are not what they seem, when we trust this illusion the choice made is hard to accept and it is hard for others to reason how we came to that decision. This is the theme that we see in the dialogue between Hamlet and his mother Gertrude in the III, iv, ll. 63-117. This theme is shown through the use of diction, imagery, repetition, and characterization. In this dialogue it is both Gertrude and Hamlet that speak to each other. At this point hamlet criticizes her strongly about Claudius and her sense of judgment. The event that has just occurred preceding this dialogue is the murder of Polonius by Hamlet- as he mistook him for Claudius. At this point in the play Hamlet knows that Claudius has killed his father and is now sure he wants to avenge him. What happens after this dialogue is that the ghost of Hamlet’s father appears to remind him of his mission, Gertrude is unable to see the ghost but Hamlet goes on to explain to her everything after the ghost goes away. The first techniques to be discussed are imagery and diction and their role in portraying the first part of the theme, that being â€Å"often times things are not what they seem†. The first line of the passage says: â€Å"the counterfeit presentment of two brothers† in reference to Hamlet’s father and Claudius. From the very beginning of the passage the sub-theme of deception of perception is brought about. Hamlet goes on to degrade Claudius by use of insults (made up of images, in turn made up of diction). â€Å"A murderer and a villain; A slave that is not twentieth part of the tithe Of your precedent lord; a vice of kings; A cutpurse of the empire and the rule, That from a shelf the precious diadem stole, And put it in his pocket! † In this quote Hamlet is unveiling the true nature of Claudius, especially by treating him of a murderer and a cutpurse (pickpocket). These two insults are references to the murder of Hamlet’s father and the thievery of the crown; two crimes that prove to be very deceiving of a king. Once Hamlet had explained this to Gertrude she realized her wrong choice. At this point we can characterize Gertrude as being repressive and denying. This is shown through her diction and imagery; â€Å"O Hamlet, speak no more:Thou turn’st mine eyes into my very soul; And there I see such black and grained spots As will not leave their tinct. † Her demand of Hamlet to cease his attacks show that she is unable to face the truth, she goes on to say â€Å"these words, like daggers, enter in mine ears† which proves further that the truth about her choice is too painful for her to admit. The image of the daggers in conjunction with her â€Å"black and grained spots† bring about the omnipresent theme of order versus disorder- which can apply in this situation as Gertrude is in a disorderly situation, where she cannot accept the truth. Another technique that shows her fear of the truth is repetition. â€Å"No more† is repeated three times as she begs hamlet to stop his attacks, showing an increasingly greater desire to avoid the truth. This fits in to the main theme as she clearly shows that it is hard to accept the wrong choice that she has made. Finally the last aspect of the main theme of the passage- how it is difficult to reason a bad choice- is brought up by hamlet more so than by his mother. In the beginning of the passage he goes on to question her choice of lower quality of man, comparing his father to god’s and later referring to Claudius as a criminal. â€Å"See, what a grace was seated on his brow; Hyperion’s curls; the front of Jove himself; An eye like Mars, to threaten and command; A station like the herald Mercury† All the references are to male gods of great importance and strength such as Mars that is god of war, Hyperion that is god of the sun (as is Jove) and finally Mercury the messenger god, and god of commerce. He goes to say that this husband was great but now she is married to a â€Å"mildew’d ear†- an image that is degrading to Claudius. â€Å"†¦ have you eyes? †¦ Nor sense to ecstasy was ne’er so thrall’d† In this long quote Hamlet goes to question what could’ve possibly given his mother to marry this man. Without giving his mother the opportunity to explain herself, he cancels out the possibility of love, as she is too old to feel the excitation and passion of love. This fits in the theme as Hamlet shows that he is unable to understand Gertrude’s decision, as for him it lacks reasoning and doesn’t offer her a chance to explain herself. Through this text analysis we can see how the theme of how often times things are not what they seem, when we trust this illusion the choice made is hard to accept and it is hard for others to reason how we came to that decision. This theme represents the situation that Hamlet was in as he was by himself in having to find out if the ghost was real or unreal, and if it was evil or good- it is the situation he was in when he had to act so strangely to the whole exterior world, when inside he knew he was sane. At this point in the play hamlet is wrong to attack his mother in such a way that is why the ghost of Hamlet’s father comes at the end of the scene to remind him of the real mission. He let his private griefs get to him. After his father’s visit Hamlet makes his mother a confident and becomes more trusting towards her. She on the other hand thinks her son is delusional. In conclusion everyone has their reason to make choices which cannot be comprehended by others, and they may have negative ramifications.